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| syndrome, strains and sprains, fatty streak , ban (law), oralhealth, wiley, cla, mayoclinic, acid, polyunsaturates, nerve, fat girls porn , obesity, tooth, | She again experienced marked reduction in pelvic pain. Comment The plasma phospholipids of this patient. reflecting hepatic EFA metabolism, reveal a block in D6DH activity affecting both n6 living and n3 EFA families. living The consequences of this block are the opposite of those in Case 1. Impaired LA desaturation produced no symptomatic clinical effect. Impaired LNA desaturation was apparently associated with pain, probably PG-mediated, that responded to LNA administration. This suggests that one physiological role of n3 fatty acids is the regulation of living n6 metabolism and PG synthesis. A superior response to LSO compared to Max-EPA in this case is unexpected and might reflect the higher dose of LSO employed. Case 3 A 55-year-old man presented for evaluation of chest pain. Three years prior to this evaluation. during a routine physical examination. |
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| Abdominal. pelvic, and rectal examinations were normal. Because dysmenorrhea is associated with increased fatty streak production fatty streak of PGF2. from AA [66], supplementation was started with 15 gm/day of linseed oil (LSO), which contains 50% LNA. During fatty streak the first month of therapy she experienced a 90% reduction in pain. After 3 months she became nauseated by LSO and was unable to swallow it. With discontinuation of LSO, her pain returned. EPO was ineffective in preventing pain but did not exacerbate it. MaxEPA, 3 gm daily, was tolerated by the patient but was not as effective as LSO. After 4 months of experimentation with various fatty acid supplements, the patient returned to the use of 15 gm LSO per day and improved her tolerance by mixing it with food. |
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