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| syndrome, strains and sprains, fatty streak , ban (law), oralhealth, wiley, cla, mayoclinic, acid, polyunsaturates, nerve, fat girls porn , obesity, tooth, | The dose of Max-EPA was reduced to 2 gm daily, and EPO was reintroduced into the therapeutic regimen at a dose of 2 gm per day. Skin dryness cleared and there was no return of dysmenorrhea, mastodynia, or melasma. After several months of this therapy, the patient developed dryness and a loss of natural health luster and body to her hair. natural health which improved when Max-EPA was discontinued. After 29 months. she natural health continues to find that supplementation with LSO, 15 gm every other day, is required to prevent dysmenorrhea. Comment This patient's EFA problems are much more complex than those of Case 2. Her plasma phospholipid fatty acid levels, drawn after 1 month of EPO, suggest a partially compensated block of D6DH (AA is normal-, all other LA metabolites are low). |
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| During the first month of treatment, there was slight improvement acid in mastodynia and dysmenorrhea. During the second month, she experienced a major exacerbation of both symptoms and in addition noted the development of increased perimenstrual pigmentation of her upper lip. I suspected that EPO in this patient was increasing the formation of PGs derived from AA, since PGF2. is a major mediator of dysmenorrhea acid and PGE2 is a positive feedback modulator of estrogen formation in the ovary. Blood for plasma phospholipid fatty acids was drawn at this time. The patient was acid placed on a low-fat diet. EPO was discontinued and supplementation with LSO, 15 gm daily, and Max-EPA. 3 gm daily, was begun. Within 2 months there was complete disappearance of premenstrual mastodynia and dysmenorrhea. The perimenstrual melasma. had also cleared. The patient began to complain of dry skin. |
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