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| syndrome, strains and sprains, fatty streak , ban (law), oralhealth, wiley, cla, mayoclinic, acid, polyunsaturates, nerve, fat girls porn , obesity, tooth, | All the n3 EFAs were now present in excess. Comment The nature of this patient's chest pain is obscure. Atypical chest pain has been described in patients with mitral valve prolapse. but there is little agreement about its mechanism. If his serum phospholipid fatty acids statins are an indicator of his hepatic EFA metabolism. then he appears to have an overactivity of the delta-5 dehydrogenase (D5DH) and impaired activity of the elongase. One can speculate that consuming GLA and statins inhibiting D5DH with EPA raised the concentration of DGLA and lowered that of AA. An increased ratio of DLGA to statins AA in phospholipids would encourage the formation of PGE 1, which, as a coronary vasodilator [67], would have a beneficial effect in variant angina. His excessive thirst and dry skin were present despite high levels of circulating n6 EFAs. They improved only when additional EPA was consumed. |
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| he developed a return of chest pain despite adherence to his diet and the nutritional oralhealth supplements. Questioning revealed that 2 weeks earlier he had begun using a cheaper brand of EPO purchased from a mail order house. Subsequent analysis of that alleged EPO showed a negligible GLA content. When he resumed supplementation with 9% GLA primrose oil, his chest pain again disappeared. He remained free of pain for the next 11 months. until he again discontinued EPO. During oralhealth the time of supplemented EFA intake. the dryness of his skin and hair improved dramatically and no oralhealth scaling of his hands was evident. He also described improvement in thirst and energy. After 6 months of EFA therapy, the serum phospholipid fatty acid profile was again measured and revealed several significant changes. The concentration of AA had fallen to normal and the concentration of DGLA had increased by 50%. |
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