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| cold sores, saw palmetto, rheumatism, hardcore galleries plump rumps , big n plump , irritable, echinacea, media, fat ass white girls , vinpocetine, | The blood is the carrier of triacylglycerols in trans fatty acids the form of VLDLs and chylomicrons, fatty acids bound to albumin, trans fatty acids amino acids, lactate, ketone bodies and glucose. The pancreas is the primary organ involved in sensing the organisms dietary and energetic states via glucose concentrations in the blood. In response to low blood glucose, glucagon is secreted, whereas, in response to elevated blood glucose insulin is secreted. The regulation of fat metabolism occurs via two distinct mechanisms. One is short term regulation which is regulation effected by events such as substrate availability, allosteric effectors and/or enzyme modification. ACC trans fatty acids is the rate limiting (committed) step in fatty acid synthesis. |
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| The resultant oxaloacetate is converted to malate by malate dehydrogenase (MDH). Pathway for the movement of acetyl-CoA units from within the mitochondrion to the cytoplasm for use in lipid and cholesterol biosynthesis. Note that the cytoplasmic malic enzyme catalyzed reaction generates NADPH which can be used for reductive biosynthetic reactions such as irritable those of fatty acid and cholesterol synthesis. The malate produced by this pathway can undergo oxidative decarboxylation by malic enzyme. The irritable co-enzyme for this reaction is NADP+ generating NADPH. The advantage of this series of reactions for converting mitochondrial acetyl-CoA into cytoplasmic acetyl-CoA is irritable that the NADPH produced by the malic enzyme reaction can be a major source of reducing co-factor for the fatty acid synthase activities. back to the top Regulation of Fatty Acid Metabolism One must consider the global organismal energy requirements in order to effectively understand how the synthesis and degradation of fats (and also carbohydrates) needs to be exquisitely regulated. |
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