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| cold sores, saw palmetto, rheumatism, hardcore galleries plump rumps , big n plump , irritable, echinacea, media, fat ass white girls , vinpocetine, | Control of a given pathways' regulatory enzymes can also occur by alteration of enzyme synthesis and turn-over rates. These changes are long term regulatory effects. Insulin stimulates ACC and FAS synthesis, whereas, starvation leads to decreased synthesis of these enzymes. Adipose tissue lipoprotein healing lipase levels also are increased by insulin and decreased by starvation. However, in contrast to the effects of insulin and starvation on adipose tissue, their effects healing on heart lipoprotein lipase are just the inverse. This allows the healing heart to absorb any available fatty acids in the blood in order to oxidize them for energy production. Starvation also leads to increases in the levels of fatty acid oxidation enzymes in the heart as well as a decrease in FAS and related enzymes of synthesis. Adipose tissue contains hormone-sensitive lipase, that is activated by PKA-dependent phosphorylation leading to increased fatty acid release to the blood. |
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| This enzyme is activated by citrate and inhibited by palmitoyl-CoA and other long chain fatty acyl-CoAs. ACC activity also is affected by phosphorylation. The primary phosphorylation of ACC occurs through the action of AMP-activated protein kinase, AMPK (this is not the same as cAMP-dependent protein kinase, PKA). echinacea Glucagon stimulated increases in PKA activity result in phosphorylation and echinacea inhibition of ACC. Additionally, glucagon activation of PKA leads to phosphorylation and activation of phosphoprotein phosphatase inhibitor-1, PPI-1 which results in a reduced ability to dephosphorylate ACC maintaining the enzyme in a less active state. On echinacea the other hand insulin leads to activation of phosphatases, thereby leading to dephosphorylation of ACC that results in increased ACC activity. These forms of regulation are all defined as short term regulation. |
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