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| cold sores, saw palmetto, rheumatism, hardcore galleries plump rumps , big n plump , irritable, echinacea, media, fat ass white girls , vinpocetine, | With respect to fatty acid metabolism this yields dephosphorylated and inactive hormone sensitive lipase. Insulin also stimulates certain phosphorylation events. This occurs plump ass through activation of several cAMP-independent kinases. Insulin stimulated phosphorylation of ACC activates this enzyme. Regulation of fat metabolism also occurs through malonyl-CoA induced inhibition of carnitine acyltransferase I. This functions to prevent the newly synthesized fatty acids from entering the mitochondria and being oxidized. back to the top Elongation and Desaturation The fatty plump ass acid product released from FAS is palmitate (via the action of palmitoyl thioesterase) which is a 16:0 fatty acid, i.e. 16 carbons and no sites of unsaturation. Elongation and unsaturation of fatty acids occurs in both the mitochondria and endoplasmic reticulum (microsomal membranes). |
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| The activity of hormone-sensitive lipase is also affected positively through the action of AMPK. Both of these effects lead to increased fatty acid oxidation in other tissues such as muscle and liver. In the liver the net result (due to increased acetyl-CoA levels) is the production media of ketone bodies. This would occur under conditions where insufficient media carbohydrate stores and gluconeogenic precursors were available in liver for increased glucose production. The increased fatty acid availability in response to glucagon or epinephrine is assured of being completely oxidized since both PKA and AMPK also phosphorylate (and as a result inhibits) ACC, thus inhibiting fatty acid synthesis. Insulin, on the other hand, has the opposite effect to glucagon and epi leading to increased glycogen and triacylglyceride synthesis. One of the many effects of insulin is to lower cAMP levels which leads to increased dephosphorylation through the enhanced activity of protein phosphatases such as PP-1. |
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