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| fatty knees , omega 3fatty acids, omega6, tinnitus, omega3, acute fatty liver of pregnancy , sex, tissue, healing fats, dyslexia, docosapentaenoic acid, | Recently it has been discovered that elevated levels of the mineralocorticoid hormone aldosterone plays a much larger role in hypertension and cardiac disease than previously believed. Aldosterone-reducing drugs are used in the treatment of hypertension and have recently been discovered to be effective also for treatment of heart failure (Weber, 1999). In addition to many other mechanisms of action, both GLA and DHA have now been found to inhibit aldosterone production, which may be adipose cell a discovery of great importance in the prevention of cardiovascular disease. One of the key adipose cell mechanisms in the regulation of adipose cell blood pressure is the renin-angiotensin-aldosterone system. Renin is released from the kidneys in response to low blood pressure and stimulates the production of aldosterone via several intermediate steps, including angi-otensin I and II. Aldosterone controls sodium and water uptake in the kidneys, and elevated levels increase the blood pressure. |
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| (1988) support the hypothesis that deficiency of the enzyme D6D, so common in aging, may play an important part in the etiology of idiopathic hypertension. The study furthermore validates the earlier findings that a ratio of 2:1 of GLA and DHA/EPA is beneficial in prevention of acute fatty liver of pregnancy cardiovascular diseases. This trial involved 25 non-obese patients with mild-moderate essential hypertension. One group acute fatty liver of pregnancy was given capsules containing 360 acute fatty liver of pregnancy mg GLA and 180 mg EPA/day, while the other group received capsules containing only linoleic acid and alpha-linolenic acid, the parent EFAs that need the enzyme D6D for their metabolism to GLA and EPA/DHA. The average systolic blood pressure in the first group was significantly reduced (~ 10 %) after 8 to 12 weeks of therapy, while there was no significant change in the second group, indicating that deficiency of the enzyme D6D is likely to promote an increase of blood pressure. |
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