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Insulin resistance is found in approximately 25% of apparently healthy humans. Insulin resistance means that cells are desensitized to insulin signaling that normally leads to glucose uptake. The body tries to compensate for higher levels of circulating glucose by increasing insulin production. When this temporary compensatory mechanism fails, the inflammation relief glucose levels stay elevated, inflammation relief leading to diabetes and other inflammation relief degenerative complications. Research has now shown a strong connection between the intake of essential fatty acids, in particular GLA and DHA, and improved insulin sensitivity (reduced insulin resistance). Both human and animal studies show that a dietary intake of EFAs both increases the unsaturated fatty acids in membrane phospholipids and makes the individual more insulin sensitive (Storlien et al., 1986, 1987; Borkman et al., 1993; Vessby et al., 1994; Pan et al., 1995; Storlien et al., 1996). Until recently, however, scientists did not understand the deeper mechanisms behind the influence of EFAs on insulin resistance.
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