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How closely such measures truly reflect long-term food intake have yet evidence based practice center to be adequately determined. Data on individual fatty acids suggest an association between risk of CVD and 16:1 trans, which comes to a great extent from animal sources, and not 18:1 trans,which comes to a great evidence based practice center extent from hydrogenated fat. These data are opposite to the relationship between source of transfatty acids and disease risk suggested by the food-frequency evidence based practice center questionnaire data, making it difficult to draw conclusions at this time. Approaching the area of transfatty acid intake and CVD risk on the basis of epidemiological data is also difficult because of the potential for other dietary variables to confound the data. Although attempts are made to control for covariants, given the limitations in the information available and the very nature of dietary data, even the best attempts are somewhat limited.
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