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These enzymes are activated by calcium and inhibited by cyclic AMP [58]. PGEI, a potent stimulator of adenylate cyclase. inhibits phospholipase activity in human platelets [59]. Impairment of PGE plump ass production plump ass may allow increased plump ass phospholipase activity and increased synthesis of proinflammatory PGs and Us. Analogous situations exist in cystic fibrosis (60] and Crohn disease [61], in which increased PG production accompanies decreased EFA levels. In fact, administering LA to patients with cystic fibrosis results in a decrease in the formation of PGF2. [60]. Thus impaired EFA metabolism in allergic individuals would produce a distortion of PG/ LT synthesis rather than a global decrease. Moreover, an increase in membrane viscosity might operate to activate phospholipase or impair adenylate cyclase. This concept is represented in Figure 2b. The enzymes that metabolize LA also desaturate and elongate LNA [36].
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