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and in each case the pretreatment analysis of phospholipid EFAs suggested an abnormality health/fitness in EFA intake or metabolism. It is possible that these varied abnormalities in EFA levels derive from the impairment in D6DH activity that health/fitness has been described in atopic eczema and that might be a common feature of atopics. The relative affinity of PUFAsynthetic enzymes for n3 EFAs. the level of n3s in the diet, and genetic or acquired variations in EFA transport modify the biochemical and physiological manifestations of the D6DH block. The preliminary data presented in this report argue health/fitness for further studies of EFA metabolism in varied groups of atopic patients. Controlled trials of EFA therapy should consider the following propositions: 1) EFA supplementation might alter numerous symptoms and signs. not only those under study. 2) EFA supplementation might have a long-term immunomodulatory effect. 3) The relative consumption of n3 and n6 EFAs might be as important as the absolute level of consumption; some individuals show a therapeutic response to n6 supplements and others show a similar response to n3 supplements.
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