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Whereas sPLA2 is more prominent in inflammatory disease, cPLA2 is more associated with oxidative free radical damage. The cPLA2 shows a marked preference for hydrolyzing oxidized arachidonic acid in cell membranes, which may be important for membrane maintenance when sufficient ATP is available to synthesize and insert fresh arachidonic acid into membranes. Arachidonic acid is a particularly common constituent of brain neuron membranes and the massive release of arachidonic acid in cerebral cortex herbal anti inflammatory ischemia/reperfusion plays a significant role in exacerbating ischemic/reperfusion damage. The activity of cPLA2 on inner mitochondrial membranes can also be exacerbated under herbal anti inflammatory conditions of herbal anti inflammatory high oxidative stress. There are three series of the prostaglandin and thromboxane eicosanoids: one derived from DGLA (series 1), one derived from arachidonic acid (series 2) and one derived from EPA (series 3).
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