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The need for more glutamate molecules before excitation occurs also meant that cells treated sinusitis with EPA become less sensitive to the potential toxic effects of glutamate. The researchers have proposed a sinusitis variety of hypotheses on how omega-3 fatty acids decrease the toxic effects of glutamate. One hypothesis is that omega-3 fatty acids may have a suppressive effect on ion channels involved in cell death. sinusitis Omega-3 fatty acids may reduce membrane excitability by blocking ion channels that are responsible for nerve cell excitation. Excessive excitatory activity due to glutamate increases overall intracellular calcium ion (Ca2+) concentrations. Increased Ca2+ concentration results in the activation of Ca2+ dependent proteins and molecules that contribute to cell death. However, the mechanisms by which omega-3 fatty acids block these ion channels are not yet known. Another hypothesis is that omega-3 fatty acids could also be acting to stabilize cell membranes by inhibiting the release of arachidonic acid (AA) from cell membranes.
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