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but the 22-carbon polyunsaturates are markedly depressed. The n3 fatty acids show no consistent pattern but tend to be lower in plasma and red cells in atopic patients than in controls. These results are not identical to those obtained in eczema: they suggest that diminished AA availability is not the critical link between EFAs and allergy. Two aspects of the data were subjected to closer scrutiny: 1) the discrepancy between the concentration of AA and the eating concentrations of other LA metabolites and 2) the discrepancy between the plasma and erythrocyte EFA profiles. The eating distribution of plasma AA concentrations in the patient group follows a normal Gaussian curve and is inversely related to the LA concentration. Although it is possible that LA displaces AA from incorporation into plasma phospholipids, a large population study has shown a positive correlation between LA and AA levels [35].
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