One of the key audience: medical/academic researchers fat

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gabe mirkin, nutrition/agriculturenews, heart disease, artery damaging lipoprotein, information, crohns disease, organic, unsaturated fatty acids / health aspects, fatty acid, dinner recipe, homeopathy, fish oils, mirkin, asthma/respiratorynews, cell based assay, health supplements, drying oil, health & fitness / nutrition, chronic, omega minus three fatty acids , fat, high blood pressure, trans, Characteristic features of primary aldosteronism are high aldosterone levels, low renin levels and a high aldosterone/renin ratio. Since audience: medical/academic researchers elevated aldosterone audience: medical/academic researchers levels have been found to play such a major role in hypertension, a research team under Marguerite Engler investigated the relationship between GLA, DHA and aldosterone in animals. In audience: medical/academic researchers several previous experimental studies Engler et al. had demonstrated that borage oil and DHA have blood pressure lowering effects in hypertensive, normotensive, old and young rats (Engler et al., 1992, 1993). When the scientists studied the effects of GLA and DHA on the renin-angiotensin-aldosterone system in rats, genetically programmed for hypertension (Engler et al., 1998 and 1999), it became clear that both these EFAs affect the renin-angiotensin-aldosterone system.
One of the key mechanisms in the regulation of blood pressure is the renin-angiotensin-aldosterone system. Renin is released from the kidneys in response to low blood pressure and stimulates the production of aldosterone via several intermediate steps, including angi-otensin I and II. Aldosterone controls sodium and water uptake in the kidneys, and fat elevated levels increase the blood pressure. Not all cases of hypertension, however, are triggered through the renin-angiotensin system. So-called fat primary aldosteronism has traditionally been regarded as a rare cause of hypertension (1%) but has recently been discovered to be more frequent than earlier believed (10% to 15% fat of patients with essential hypertension) (Fardella et al., 1999). This condition is characterized by excess production of aldosterone in the adrenal gland, without involvement of the renin-angiotensin-aldosterone system.
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