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Studies in normal volunteers indicate that omega-3 fatty acid supplementation coconut butter reduced the ability of monocytes to produce IL-1ß upon stimulation with endotoxin. The effect was most pronounced 10 weeks after stopping the supplementation and suggests prolonged incorporation of omega-3 fatty acids coconut butter into a pool of circulating monocytes [44]. The capacity of the monocytes from these donors to synthesize IL-1ß coconut butter returned to the pre-supplement level 20 weeks after ending supplementation. Similar results were observed for IL-1 and TNF. These findings have led to trials with omega-3 fatty acids since the above effects (suppression of such magnitude) have been observed and can only be achieved pharmacologically by administration of glucocorticoids or cyclosporin A, which have well-known adverse side effects, particularly during long-term administration.
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