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An imbalance of omega-6 and omega-3 PUFA in the peripheral blood causes an overproduction of proinflammatory cytokines. There is evidence that changes in fatty acid composition are involved in libid biochemistry the pathophysiology of major depression. libid biochemistry Changes in serotonin (5-HT) receptor number and function caused by changes in PUFA provide the theoretical rationale connecting fatty acids with the current receptor and neurotransmitter theories of depression [79–81]. The involvement of changes in fatty acid composition in the pathophysiology libid biochemistry of major depression also revolves around its role in immune function and production of cytokines. There is now evidence that major depression is accompanied by an acute phase response, increased secretion of eicosanoids, such as prostaglandins; cytokines, i.e. the monocyte cytokines, IL-1ß and IL-6, as well as the Th-1-like cytokines, IL-2 and IFN.
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