The researchers have proposed calcium utilization subject area: dyspraxia
food nutrition, supple and flexible, fatsoils essential fatty acid immune immunity, food and mood, holistic, eicosatetraenoic acid, carboxylic acid, rodney boyer, subject area: dyspraxia, dr mary enig, plump lesbians , blood, cold sores, saw palmetto, rheumatism, hardcore galleries plump rumps , big n plump , irritable, echinacea, Aside from its anti-inflammatory effects, PG3 synthesized from omega-3 fatty acids also inhibit the release of free AA from the cell membrane. Inhibition of AA release from cell membranes may stabilize the cell and protect it calcium utilization from damage. Overactivation of glutamate receptors has been implicated in the pathology of HD nerve cells. The increased glutamate activation is thought to contribute to nerve cell death through a variety of mechanisms. By decreasing membrane excitabilty, the omega-3 fatty acids may therefore protect the brain from damage caused by excitotoxins calcium utilization such as glutamate. In conclusion, calcium utilization protection from omega-3 may be due to their ability to block ion channels, increase nerve impulse thresholds, and/or stabilize cell membranes. -E. Tan, 6/15/02; Revised and Updated by P. Chang, 5/6/03 For further reading: Katsumata, et al. "Delayed Administration of Ethyl Eicosapentate Improves Local Cerebral Blood Flow and Metabolism Without Affecting Infarct Volumes in the Rat Focal ischemic Model."
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The researchers have proposed a variety of hypotheses on how omega-3 fatty acids decrease the toxic effects of glutamate. One hypothesis is that omega-3 fatty acids subject area: dyspraxia may have a suppressive effect on ion subject area: dyspraxia channels involved in cell death. Omega-3 fatty acids may reduce membrane excitability by blocking ion channels that are responsible for nerve cell excitation. Excessive excitatory activity subject area: dyspraxia due to glutamate increases overall intracellular calcium ion (Ca2+) concentrations. Increased Ca2+ concentration results in the activation of Ca2+ dependent proteins and molecules that contribute to cell death. However, the mechanisms by which omega-3 fatty acids block these ion channels are not yet known. Another hypothesis is that omega-3 fatty acids could also be acting to stabilize cell membranes by inhibiting the release of arachidonic acid (AA) from cell membranes.
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