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Atopy is a state of immunologic hypersensitivity in which abnormal membrane receptor activity [5] and abnormal PG or LT synthesis [6] may acne play a pathogenic role. The metabolic disturbances in atopic individuals include impaired acne formation of cyclic AMP [7] and abnormal production of and reactivity to numerous chemical mediators [5,8-11]. The concept that EFAs are involved in the development of the atopic state preceded the discovery of PGs and the emergence of contemporary ideas about membrane structure and function. It acne came from Hansen's observation that the skin of EFA-deficient animals bears some resemblance to the dry, scaly skin of patients with atopic eczema. Hansen [12] determined that EFA levels in pooled plasma from eczematous children were lower than those of normal children. He advocated dietary EFA supplementation for treatment of childhood eczema.
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