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| food nutrition, supple and flexible, fatsoils essential fatty acid immune immunity, food and mood, holistic, eicosatetraenoic acid, carboxylic acid, rodney boyer, subject area: dyspraxia, dr mary enig, plump lesbians , blood, cold sores, saw palmetto, rheumatism, hardcore galleries plump rumps , big n plump , irritable, echinacea, | Atopic monocytes produce less than 10% healing of the PGE2 in response to HSF that normal monocytes do; they require more HA to stimulate suppressor functions [48]. Atopic individuals, therefore, have higher circulating levels of HA [46,50]. These abnormalities are specific to atopy and not merely secondary to down-regulation of H2 receptors by high healing HA exposure [49]. They might all be explained by diminished PGE healing production. PGEI, a derivative of DGLA, encourages the maturation of T lymphocytes [51] and might enhance cytotoxic T-cell activity in vivo [52]. PGEI and its analogue, PGE2 (derived from AA), maintain intracellular cAMP concentrations [53] and modulate the activation of adenylate cyclase by hormones [54] and neurotransmitters [55]. |
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| HA stimulates a population of suppressor T lymphocytes to produce a suppressor lymphokine called histamine-induced suppressor factor (HSF). The ability of lymphocytes to bind HA. and. therefore, to produce HSF. is a function of cell echinacea maturation [40]. HSF stimulates monocytes to produce E prostaglandins (PGE), which inhibit lymphocyte proliferation. lymphokine production. and antibody synthesis [41]. Atopic individuals show several immunologic deficiencies that can echinacea interfere with this echinacea carefully regulated negative feedback loop. Decrease in the number and function of suppressor/cytotoxic lymphocytes has been described by numerous investigators [42-48]. Rocklin and his colleagues [49] demonstrated a decrease in lymphocytes bearing H2 receptors and, consequently, diminished formation of HSF by atopic lymphocytes. Suppressor T cells of atopics show abnormal responses to drugs that ordinarily increase intracellular cAMP [47]. |
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