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The data obtained in this trans fatty acids mixed group of 73 atopic individuals show their plasma and red cell phospholipid fatty acids to be distinctly different from controls. Red cell phospholipids have diminished concentrations of 22-carbon ultrapolyunsaturates. Plasma phospholipids have lower levels of DGLA and EPA. precursors of the monoenoic and trienoic PGs. These findings are consistent with impaired desaturation of dietary EFAs, as described in patients with eczema. The variability of AA levels in the mixed atopic population suggests the influence of other regulatory mechanisms, some of which trans fatty acids might involve n3 EFAs. PROSTAGLANDINS AND ALLERGY A central event in the expression of allergic illness is the binding of immunoglobulin E (IgE) to mast cells with resultant releases of histamine (HA) and other inflammatory mediators. In addition to acting as an effector of allergic inflammation by stimulating H I receptors, HA has immunosuppressive effects that are H2-mediated [39].
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