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They must huntington's extract 20-carbon EFAs for PG synthesis from plasma, an important transport step catalyzed by a specific arachidonoyl-CoA synthetase [33]. EPA is a potent inhibitor of AA transport into these cells. The n3 EFAs, therefore, exert multiple regulatory effects on n6 metabolism. English investigators administered evening primrose oil (EPO) as a dietary supplement to patients whose EFA levels they huntington's studied [34]. Primrose oil was chosen because huntington's it contains 9% gamma linolenic acid (GLA. 18:3 n6), the product of delta-6 desaturation of LA. In a double-blind study using three levels of primrose oil (2.0. 4.0, and 6.0 gm daily) they found a significant, dose-dependent reduction in inflammation determined by pruritus, erythema, and scaling. EFA levels showed significant improvement as well [24]. This suggested that the intake of GLA was exerting not only a corrective effect on the usual skin signs of EFA deficiency. but an antiinflammatory and immunomodulatory effect.
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